Hemostasis Today

February, 2026
February 2026
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Sarah Mount: Pregnancy, Hypercoagulability, and Erythritol
Feb 19, 2026, 13:36

Sarah Mount: Pregnancy, Hypercoagulability, and Erythritol

Sarah Mount, Postdoctoral Researcher at Maastricht University, shared on LinkedIn:

”Pregnancy, Hypercoagulability, and Erythritol — A Conversation We Should Be Having

Pregnancy is not a neutral physiological state when it comes to clotting.

It is a well-established hypercoagulable condition, designed to protect against haemorrhage — but one that increases the risk of thrombosis.

That baseline risk is compounded by:

  • Gestational diabetes
  • Hypertensive disorders of pregnancy (including pre-eclampsia)
  • Chronic hypertension
  • Obesity and metabolic dysfunction
  • Inherited thrombophilia’s ex. Factor V Leiden (often undiagnosed, as universal screening is not routine).

Placental function depends on stable microvascular blood flow. When thrombotic risk rises, placental complications — including abruption and infarction — can escalate rapidly.

Now consider emerging data on erythritol (E968).

A 2023 study published in Nature Medicine reported that higher circulating erythritol levels were associated with increased risk of major adverse cardiovascular events (MI, stroke, death) over three years in high-risk populations.

Experimental components of the study demonstrated enhanced platelet activation and thrombosis formation in vitro and in animal models.

A subsequent human intervention study showed that ingestion of erythritol-sweetened beverages can produce plasma levels associated with these pro-thrombotic effects.

Moreover, there are more recent studies examining erythritol and blood-brain barrier integrity, as well as cardiovascular event risks.

Importantly:
These studies show association and mechanistic plausibility — not proven causation.

Erythritol remains ‘generally recognised as safe’ under current regulatory frameworks. Pregnancy-specific outcome data are lacking.

But here is the clinical question:

In a population already in a hypercoagulable state — sometimes with additional metabolic, hypertensive, or genetic risk factors — does it seem unreasonable to at least discuss minimising exposure to compounds with emerging pro-thrombotic signals?

This is not alarmism. It is risk stratification.

At minimum, this appears to be a reasonable topic for informed discussion between pregnant patients and their obstetric providers.

And then there is labelling…”

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